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Our results provide a possible mechanism in which upregulation of phosphorylated p53 and p16 maintain the cell cycle arrest of keloid fibroblasts, and FOXO4-DRI selectively induces apoptosis of keloid senescent fibroblasts by prompting p53-pS15 nuclear exclusion

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Besides producing insulin, -cells sense glucose levels and release insulin to maintain these levels within the normal physiological range (Jeffrey et al., 2010)