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The key pathological hallmarks of AD include amyloid-beta (A) plaque accumulation, neurofibrillary tangles of hyperphosphorylated tau, chronic neuroinflammation, oxidative stress, and mitochondrial dysfunction (Lv et al., 2020)

RBC membrane is an important target for both endogenous and exogenous ROS that may induce oxidative changes in membrane proteins and lipids