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On the contrary, high numbers of invading bacteria exert an overactivation of Toll-like receptors (TLRs), leading to the overexpression of inflammatory cytokines, with consequent epithelial damage and chronic inflammation [10]

The exact minimum effective concentration that recapitulates our observed suppressive ILC2 phenotype in vivo , remains to be determined, and will likely rely on a complex grouping of factors that take into account systems with endogenous melatonin rhythms (such that were absent in our studies both in vitro and in vivo with BALBc mice)

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