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The problem: very short half-life (~50 minutes) and 3 daily injections at meals , an extremely impractical regimen

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It has been argued, in the broad context of synaptic plasticity at excitatory synapses, that an emphasis on [Ca 2+ ] i rise as the triggering mechanism of plasticity can offer improved explanatory value over a fixation on learning rules, such as STDP (Lisman and Spruston, 2+ ] i rise is strong local activation and local spread to inactive synapses or more global influx through voltage gated channels activated by back-propogating action potentials and amplified by release from internal stores

On the neuroprotective role of astaxanthin: New perspectives