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This positions GHK-Cu as a model substrate for studying small-peptide control of large gene-network programs.[9] Redox-Silenced Copper Delivery Studies Bioinorganic chemistry investigations use GHK-Cu as a model carrier for delivering Cu(II) to cells without triggering Fenton-driven hydroxyl-radical generation

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Decreased NAD + levels can contribute to sirtuin mediated deacetylase-dependent alterations in both mitochondrial electron transport activity (resulting in increased production of reactive oxygen species, ROS) as well as to increased nuclear DNA damage leading to the activation poly-ADP-ribose polymerases (PARP)

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