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Genetics play a role in B12 deficiency Genetic polymorphisms such as MTHFR or MTRR can contribute to low usable B12

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readers can compare how a pure GLP-1 mechanism operates in the discussion of how semaglutide activates GLP-1 receptors and how dual-incretin agents such as tirzepatide engage overlapping but broader circuitry in tirzepatides influence on incretin pathways