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Antibiotics with different mechanisms of action induce distinct patterns and kinetics of membrane potential changes: membrane-targeting antibiotics (e.g., polymyxins) typically trigger rapid and sharp depolarization, protein synthesis inhibitors (e.g., kanamycin) may cause a slower, more gradual depolarization, while nucleic acid inhibitors might show little early depolarization

Natural-source vitamin E is more active in our bodies than synthetic vitamin E

[DOI] [PubMed] [Google Scholar] Franciotta D., et al

(137) further indicated that DKD microvascular complications are closely associated with abnormal BCAA metabolism, with high levels of L-leucine [area under the curve (AUC) = 0.834] and isoleucine (AUC = 0.932) demonstrating high diagnostic ability in distinguishing DKD from T2DM

doi: 10.1073/pnas.92.19.8700