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This is mechanistically demonstrated in breast cancer models, where SFN, by activating the Nrf2 pathway, reduces the secretion of prostaglandin E2 (PGE2) from tumor cells, thereby triggering MDSCs to switch from an immunosuppressive to an immunogenic phenotype and inhibiting their expansion ( Emerging evidence further indicates that SFN promotes the repolarization of tumor-associated macrophages from the protumoral M2 phenotype toward the antitumoral M1 state

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[Abstract]2025 Nov 25:259:115314

Targeted anti-tumor agents that take advantage of the unique chemical environments of cancer cells greatly reduced side effects on normal cells

Also, miR-NPs showed a significant reduction in expression of P-P65 and MEKK3 protein kinase