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Perturbations of NAD(+) salvage systems impact mitochondrial function and energy homeostasis in mouse myoblasts and intact skeletal muscle

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reported increased TNF-, VEGF, CD31, and TGF- in IC/BPS patients with and without Hunner lesions compared with controls, whereas increased mast cell tryptase and collagen were observed in Hunner lesion patients only (compared with nonHunner lesion patients and controls)