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Table 1 Human clinical studies evaluating gut microbiota changes and microbial features associated with GLP-1 receptor agonist therapy

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Concurrently, high glucose suppressed the tumor suppressor TIF1, further amplifying Smad-mediated transcription and elevating the expression of pro-oncogenic genes, including the epithelialmesenchymal transition markers N-cadherin and vimentin, thereby collectively fostering CRC progression

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