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Changes to quaternary structure may allow LPL to self-regulate as it navigates its different roles moving from an inactive form stored in adipocyte vesicles 5 , to a heterodimer on the capillary wall 8,9 , and finally onto lipoproteins as a homodimer to eventually facilitate uptake by the liver 67

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Instead of focusing solely on behavior, these treatments address underlying biological drivers

10.3390/ijms222413198 13 AlkasirR.LiJ.LiX.JinM.ZhuB