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Loss of autophagy causes marked accumulation of p62 that in turn suppresses Keap1 to activate Nrf2 and increase ARE gene expression

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The findings are interesting

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Ultimately, this FOXO4-DRI-mediated mechanism selectively eliminates senescent endothelial cells, which in turn attenuates vascular inflammation and improves endothelial-dependent vasodilation, conferring a significant protective effect on vascular function