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GLP-1 receptor agonists (in combined therapy with enzalutamide) diminish the levels of Akt and mTOR, leading to decrements in nuclear androgen receptor localization

The introduction of wortmannin was found to significantly reverse the expression of PI3K and AKT, as well as the anti-apoptotic and anti-inflammatory effects of Dihexa, resulting in a decrease in the number of neuronal cells present in the cortex and the levels of IL-10, and an increase in the levels of TNF-alpha and IL-1-beta [1]

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and Nakaki, T