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Specifically, the accumulation of oxidized low-density lipoprotein (oxLDL) in Ms enhanced LPS-induced expression of NRF2-dependent ROS detoxification enzymes (i.e., GSR) and suppressed the expression of HIF-1-dependent ROS producing enzymes (i.e., NOS2)

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fragilis- derived LPS drive NF-B, which may help promote neuroinflammation and pathogenesis during AD [333]