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Tarrag, M

Deficiency of the X-inactivation escaping gene KDM5C in clear cell renal cell carcinoma promotes tumorigenicity by reprogramming glycogen metabolism and inhibiting ferroptosis

These findings correlate the altered expression of key targets directly with potential pathogenic mechanisms and therapeutic targets of BRCA

While its roles in nuclear factor erythroid 2-related factor 2 (Nrf2) activation and histone deacetylase (HDAC) inhibition are well-established, this review provides a novel synthesis by integrating disparate research scalesa multiscale perspective that spans from the genetic and epigenetic regulation of glucoraphanin biosynthesis in plants to SFNs recently elucidated effects on ferroptosis, cancer stem cells (CSCs), and the tumor immune microenvironment in humans

The theoretical framework for synergy rests on well-established principles of systems biology