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In a PD rat model treated with 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), long-term use of EGb761 prevented the loss of dopaminergic nerve terminals caused by MPTP (Ramassamy et al., 1990)

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The 2025 paper is the most recent significant independent confirmation of the in vitro mechanistic claims about Epithalon and represents the kind of external replication that the Epithalon literature had previously lacked