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Tumours were allowed to develop for 56 weeks until mice showed signs of tumour burden, at which point mice were randomized and treated with a single i.p

Mechanistic investigations in human somatic cell cultures have suggested that Epithalon may: Activate telomerase in vitro Support telomere maintenance and reduce the accumulation of DNA replicationassociated errors Potentially modulate the balance between cellular senescence and replicative capacity in experimental systems In parallel, Epithalon has been examined for its ability to interact with oxidative stress pathways and free radical markers, which may contribute to observed longevity-related effects in certain preclinical models

J Biol Chem 279:3567135678

As limited in current reference reports, more human data and toxicological investigation is needed to further elucidate the adverse actions of prenatal APAP exposure to offspring

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