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While the phenotypic insulin resistance in NAFLD would be expected to counteract insulin-mediated SREBP1c activation, a state of selective insulin resistance ensures that insulin retains its ability to promote DNL through SREBP1c while being unable to suppress gluconeogenesis [41]

If redness persists for hours, spreads beyond the application area, or worsens over time, reduce concentration or frequency

[DOI] [PubMed] [Google Scholar] 78.Qiao J., Chen L., Huang X., Guo F

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