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Nature.com.

Cellular senescence and senolytics: the path to the clinic | Nature Medicine n.d

Methods The current study involves the fusion of a native or mutant GLP-1 (30 residues) with an HSA-binding DARPin (123 residues) via a rigid helical linker (15 residues) to create fusion proteins (168 residues) resistant to DPP-IV and, or trypsin with extended half-lives using structural modeling, which are subsequently characterized using MD simulation and molecular docking analyses

A clinical review in a peer-reviewed journal proposed exactly this: a serotonergic mechanism by which GLP-1 agonists could theoretically decrease sexual desire, and a biopsychosocial perspective on why this effect may be camouflaged by competing influences