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Chronic stress signals, including hyperglycaemia, inflammation, neurohormonal activation, and mechanical overload, promote the activation of cardiac fibroblasts and the accumulation of extracellular matrix within the myocardium

This is consistent with many animal studies and in-depth immunological studies that show, as expected, a measurable contribution to protection via non-nAb [56,57,58], NK cells [59], and MAIT cells [60, 61] and in particular T cells [62,63,64,65]

USA 102 , 91949199 (2005)

With over 20 years of clinical expertise, Dr

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