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Endocrinology 159 , 665675 (2018)

While cagrilintide has not yet been approved for therapeutic use, its mechanisms of action have inspired further exploration in research settings, particularly in preclinical and early-phase clinical studies focused on gastrointestinal and metabolic endpoints

Impaired intestinal afferent nerve satiety signalling and vagal afferent excitability in diet induced obesity in the mouse

(2025) discovered that Talaromyces purpurogenus , enriched in the gut of PCOS patients, produces the metabolite AT-C1, which suppresses the AhR signaling pathway and reduces IL-22 secretion, thereby inducing PCOS-like symptoms (69)

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