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It plays a critical role in neutralizing free radicalsunstable molecules that can cause cellular damage

Patients should maintain realistic expectations and continue treatment as prescribed, even if immediate improvements are not apparent, as premature discontinuation can lead to symptom recurrence

We demonstrated that the proposed drugs could effectively suppress glutaminase and reduce glutamate production in the diseased brain through multiple neurodegeneration-associated mechanisms, including cytoskeleton and proteostasis

Methionine uptake via the SLC43A2 transporter is essential for regulatory T-cell survival