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Extensive research across various animal models has demonstrated its ability to mitigate optic nerve damage, axon loss, and retinal vascular damage, suggesting its potential to RGCs and their axons (186, 187)
They are synthesised in excess during untreated (or inadequately treated) type 1 diabetes mellitus
After six weeks of treatment, serum selenium concentrations increased by 53 percent and, although they dropped a little, remained at 45 percent above baseline for the duration of the treatment
Hayward SW, Wang Y, Cao M, Hom YK, Zhang B, Grossfeld GD et al