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This observation aligns also with a generally lower correlation in gene expression in hypothalamic neurons of both mice treated with the GIPR antagonist or acyl-GLP-1, and further in mice treated with acyl-GIP or MAR709 (Extended Data Fig

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Demonstrates marked gastroprotective and ulcer-healing activity in rodent models of IBD, NSAID damage, and fistula formation, mirroring the protective role of the parent protein in human gastric juice is exposed to daily