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10.1016/S0140-6736(17)30182-4 79 PatrascuO.Bguet-CrespelF.MarinelliL.Le ChatelierE.AbrahamA.-L.LeclercM.et al (2017)

Research indicates the anti-fibrotic effects may be mediated through suppression of transforming growth factor beta (TGF-) signaling, a major driver of pathological cardiac fibrosis, and reduced activation of cardiac fibroblasts

In this Review, we explore how mitochondrial metabolism contributes to therapeutic resistance, with particular emphasis on metabolic plasticity, redox balance, and organelle quality control

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