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When such a magnitude of weight reduction is achieved by caloric restriction alone, increases in appetite caused by weight loss-induced modifications in neuroendocrine hormones that regulate hunger (increased ghrelin), satiety (decreased peptide YY, amylin), and fullness (decreased cholecystokinin) would be expected [66]

Importantly, this process respects the bodys natural feedback mechanisms somatostatin, the hormone that inhibits GH release, continues to function normally

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