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It was also proved that matrix-detached epithelial and cancer cells cluster spontaneously through a pathway involved with Nectin-4 (also known as cell adhesion protein PVRL4), the process of which sustains GPX4 expression and buffers against lipid peroxidation by stimulating the PVRL4/64/Src axis signal pathway (Brown et al., 2018)

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Environ Pollut 292:118390 Hou L-S, Zhang Y-W, Li H, Wang W, Huan M-L, Zhou S-Y et al (2022) The regulatory role and mechanism of autophagy in energy metabolism-related hepatic fibrosis

Overnight, the skin becomes more receptive to active ingredients, making GHK-Cu even more powerful