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Finally, we highlight the latest research developments concerning iron-regulated therapeutic agents and rational treatment strategies aimed at targeting ferroptosis

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[12] Ghrelin receptors are known for their role in controlling appetite, and their activation may lead to heightened hunger cues, potentially resulting in increased body mass

The gene silencing of ACSL4 in primary microglial cells has been reported to impede ferroptosis of microglia induced by HIV-1 Tat protein, with an effect comparable to that of the administration of Fer-1 or DFO (38)

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