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The aforementioned glutamine transporters are promising drug targets that can inhibit the uptake of glutamine by tumor cells from the source, block the nutritional and energy sources of tumor cells, and thus weaken the advantage of tumor cells in glutamine competition (Fig

Deficiency or excess of Se, I, Zn, as well as Fe and Cu affects the synthesis of TG and can disrupt thyroid homeostasis (101)

These reactions include increasing cAMP and intracellular calcium, activation of protein kinase A, and induction of gene transcription [33]

250 These mice exhibit impaired axonal mitochondrial membrane potential and bioenergetic function, indicating a lack of reserve capacity

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