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In preclinical models, SAM-e protected against amyloid-beta-induced cellular injury by inhibiting oxidative stress and neuroinflammation (34)

Collectively, these findings indicate that antioxidant and polyphenol-based compounds may have therapeutic potential in the management of FM through their modulation of oxidative stress, inflammation, and neurotransmitter homeostasis

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Overall, there is currently little evidence from RCTs that antioxidant supplementation improves memory performance in individuals with neurodegenerative conditions