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In 1990, Austin coined the term atherogenic lipoprotein profile, which describes the syndrome of small, dense LDL, elevated VLDL, and low HDL ( Figure 1 ) [21]
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of this final rule with comment period) is included in the budget neutrality calculations for the CY 2026 OPPS
[9] The mechanism appears to involve direct GHS-R1a receptor activation on cardiac cells independent of GH release, since hexarelin (a related GHRP) showed cardioprotective effects even in GH-deficient models
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