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albicans can finely regulate multiple iron uptake pathways such as high-affinity iron reduction systems and heme uptake through mechanisms such as the Sef1-Sfu1-Hap43 regulatory circuit and the iron-sulfur cluster assembly system, thereby promoting invasive and morphological transformation processes related to virulence, and accelerating the progression of infection (Zheng et al., 2025)

Twenty rare NCP1L1 alleles have been found in the low cholesterol absorbers and appear to impair NCP1-L1 cholesterol uptake through various mechanism ([47, 48]

Polarization of macrophages along the M1 and M2 axes occurs based on the activities of the arginine metabolizing enzymes NOS and arginase, respectively (Wentzel)