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In the presence of suitable precursors for GSH synthesis, cultured astrocytes can fully compensate for the exportmediated GSH loss by GSH de novo synthesis, even if Mrp1mediated GSH export was further stimulated (Arend et al
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Although these mice initially exhibit normal cardiac function, they progressively develop left ventricular dilation, reduced fractional shortening, disorganized sarcomeres, and damaged mitochondria with aging, highlighting the essential role of sustained autophagy in maintaining structural integrity and contractile function over time
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