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It points to GLP-1 receptors being stimulated throughout the body, including in the brain's appetite and nausea centers, and says that widespread activation combines with rapid changes in blood glucose and hydration status during early treatment

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In agreement with the modulation of inflammation elucidated through in silico analysis, an in vivo OVA-induced BALB/C model sought to determine if the attenuation of airway inflammation was mediated by regulation of autophagy [160]