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Sinensetin attenuates hepatic ischemia-reperfusion injury through suppressing grp78/chop-mediated endoplasmic reticulum (Er) stress in mice

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We reasoned that if decreases in glutathione and glutamate in the stable phase are a consequence of damage arising in a preceding acute phase, a sample of stable patients would be more likely to demonstrate a reduction in these neurochemicals than a more heterogeneous sample