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Interestingly, all intervention groups showed similarly reduced fasted blood glucose concentrations at study endpoint that were significantly lower than Veh (Fig

These findings underscore lipid peroxide accumulation as a hallmark of ferroptosis, and suggest that downregulating ACSL4, LPCAT3, LOXs, or POR can suppress PUFA-PL-OOH formation and effectively inhibit ferroptosis

These changes drive conversion to the pro-inflammatory M1 phenotype, which exhibits anti-tumor properties ( M2-type TAMs enhance intracellular heme accumulation by phagocytosing senescent erythrocytes

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