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Consistent with these in vivo results, vitamin K1 treatment impeded RSL3- or erastin-induced ferroptosis by not only regulating GPX4 expression but also modulating the levels of FSP1 and DHODH, two newly identified factors that defend against ferroptosis [114]

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1200 MPs/mL) significantly delayed hatching at 48 h post-fertilisation (hpf) above 50 MPs/mL, although no significant effect on embryo growth and survival and apparent deformities was observed

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