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The data presented here suggest that Nlel-AnglV and Dihexa increase miniature synaptic activity by increasing dendritic spine densities and altering the morphological phenotype of postsynaptic spines in- vitro and may represent the mechanism that underlies facilitated learning observed AnglV analogues

Actomyosin contractility can compromise nuclear integrity by squeezing the nucleus dorsoventrally [191, 192], pulling it [193] or promoting the nuclear influx of cytoplasmic constituents, which in turn pressurizes the nucleus, leading to its rupture [155]

T2DM patients exhibit a 3-fold higher susceptibility to tuberculosis, directly reflecting measurable functional failure of NK cells and T cells

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