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Bisphenol-A treatment also reduces the number of differentiating neurons from an immortalized human NSPC line known as ReNcell VM ( Recent investigations have demonstrated that bisphenol-A affects mitochondrial biology in hippocampal NSPCs, thus leading to increased oxidative stress and apoptosis (Figure 3)

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ER Stress and Unfolded Protein Response Accumulation of unfolded and misfolded proteins or excessive protein trafficking in the lumen of the ER causes ER stress, which triggers the unfolded protein response (UPR)