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Nicotine addiction, mainly smoking, is the leading cause of lung cancer and other diseases of the lungs, including chronic bronchitis and emphysema

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This review summarizes results from studies of humans, preclinical animal models, and cultured endothelial cells that document toxicities of cigarette smoke exposure on pulmonary endothelial cell functions, including barrier dysfunction, endothelial activation and inflammation, apoptosis, and vasoactive mediator production

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