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It is possible that the tar, nicotine, and carbon monoxide in cigarette smoke 40 activate ILC3s by binding to toll-like receptors and aryl hydrocarbon receptors 41

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In addition, genes involved in tight junctions formation, intraflagellar transport gene and cilia-related genes are markedly down-regulated in the airway epithelium of healthy smokers when compared with nonsmokers [131]

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